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Former Grad Student, Dr. Kim Hawkins & Candelario-Jalil lab publish in April 2017 Brain and Behavior

BACKGROUND: Resolution of inflammation is an emerging new strategy to reduce damage following ischemic stroke. Lipoxin A4 (LXA 4) is an anti-inflammatory, pro-resolution lipid mediator that reduces neuroinflammation in stroke. Since LXA 4 is rapidly inactivated, potent analogs have been synthesized, including BML-111. We hypothesized that post-ischemic, intravenous treatment with BML-111 for 1 week would provide neuroprotection and reduce neurobehavioral deficits at 4 weeks after ischemic stroke in rats. Additionally, we investigated the potential protective mechanisms of BML-111 on the post-stroke molecular and cellular profile.

Graduate Student, Kelly DeMars & Candelario-Jalil lab publish in April 2017 Journal of Experimental Neuroscience

DeMars KM, Yang C, Hawkins KE, McCrea AO, Siwarski DM, Candelario-Jalil E. Spatiotemporal Changes in P-glycoprotein Levels in Brain and Peripheral Tissues Following Ischemic Stroke in Rats. J Exp Neurosci. […]

Wolfgang Streit

Dr. Jake Streit & colleagues publish in May 4, 2017 Equine Veterinary Journal

Delcambre GH, Liu J, Streit WJ, Shaw GPJ, Vallario K, Herrington J, Wenzlow N, Barr KL, Long MT. Phenotypic characterisation of cell populations in the brains of horses experimentally infected […]

2017 Neuromedicine Summer Seminar Series

2017 Neuromedicine Summer Seminar Series

Setting Benchmarks | BMS Program Overview

Setting Benchmarks | BMS Program Overview

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